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  • Integration of lipidomics with targeted, single cell, and . . .
    This process is called lipid mediator class switching (LCS) and has been identified as an essential mechanism for resolving inflammation 10 This LCS process appears to be deficient in CRC, resulting in the majority of tumours sustaining a chronic inflammatory state In this study, we have profiled the untargeted and targeted lipid landscape and
  • Lipid signalling in disease | Nature Reviews Molecular Cell . . .
    Imbalances of these major lipid signalling pathways contribute to disease progression in chronic inflammation, autoimmunity, allergy, cancer, atherosclerosis, hypertension, heart hypertrophy
  • Role of Inflammation in the Development of Colorectal Cancer
    Subsequently, chronic inflammation, together with genetic and epigenetic changes, have been shown to lead to the development and progression of CRC Various cell types present in the colon, such as enterocytes, Paneth cells, goblet cells, and macrophages, express receptors for inflammatory cytokines and respond to tumor necrosis factor-alpha
  • Bioactive lipids, inflammation and chronic diseases
    Endogenous bioactive lipids are part of a complex network that modulates a plethora of cellular and molecular processes involved in health and disease…
  • Changes in lipids composition and metabolism in colorectal . . .
    Altered metabolism of lipids is currently considered a hallmark characteristic of many malignancies, including colorectal cancer (CRC) Lipids are a large group of metabolites that differ in terms of their fatty acid composition This review summarizes recent evidence, documenting many alterations in the content and composition of fatty acids, polar lipids, oxylipins and triacylglycerols in
  • Role of Lipid Accumulation and Inflammation in . . .
    Cellular cholesterol and inflammation can affect the immune system and autoimmune diseases Ox-LDL and generally modified lipoproteins increase endothelial damage, leukocyte recruitment, and inflammation Several studies revealed that high levels of E-selectin, ICAM-1, and VCAM-1 are expressed by inflamed endothelium





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