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  • TNFRSF11A protein expression summary - The Human Protein Atlas
    The protein encoded by this gene is a member of the TNF-receptor superfamily This receptors can interact with various T RAF family proteins, through which this receptor induces the activation of NF-kappa B and MAPK8 JNK This receptor and its ligand are important regulators of the interaction between T cells and dendritic cells
  • TUMOR NECROSIS FACTOR RECEPTOR SUPERFAMILY, MEMBER 11A; TNFRSF11A - OMIM
    Dendritic cells are rare hematopoietic cells that capture, process, and present antigens to T cells Anderson et al (1997) identified dendritic cell cDNAs encoding a protein with homology to the extracellular domain of tumor necrosis factor receptor (TNFR) superfamily members They designated the predicted 616-amino acid protein RANK, for 'receptor activator of NF-kappa-B' (see 164011)
  • POS0372 TNFRSF11A AS A CAUSATIVE GENE IN SYSTEMIC AUTOINFLAMMATORY . . .
    Background: Limited evidence suggests that TNFRSF11A as a causative gene in systemic autoinflammatory diseases (SAIDs) [1,2] Objectives: To estimate the prevalence of TNFRSF11A variants in a cohort of patients with SAIDs and describe their disease phenotypic expression Methods: Consecutive patients (n=167) with confirmed SAIDs who underwent screening by next generation sequencing targeting
  • AAIR :: Allergy, Asthma Immunology Research
    Immunohistochemistry The mouse lung tissues were fixed in 10% neutral formalin, paraffin-embedded, and cut into 5 μm-thick sections The paraffin sections were antigenically repaired, and endogenous peroxidase was removed and then blocked with 5% bovine serum albumin for 1 hour at 37°C TNFRSF11A shRNA also decreased the HDM-induced
  • How does atherosclerotic plaque become calcified, and why?
    (atherosclerotic, mostly intimal), (2) metabolic (chronic kidney disease [CKD] and diabetes mellitus [DM], affecting both intimal and medial, also known as Monckeberg’s calcification), and (3) genetic causes [1] Atherosclerotic intimal calcifications are distinct from medial calcifications, though overlapping factors can contribute to both
  • Osteo Chondrocytic Transcription Factors and Their Target Genes Exhibit . . .
    calcified plaque, nine carotid lesions were used for in situ hybridization and immunohistological analysis In these le-sions calcification invariably colocalized with lipid and al-though intimal calcification was most prevalent, in some lesions, the media beneath large atherosclerotic plaques was
  • Subcellular - TNFRSF11A - The Human Protein Atlas
    tnfrsf11a resources tissue brain single cell subcellular cancer blood cell line structure interaction about introduction history organization publications antibody submission antibody availability acknowledgments contact news news articles press room learn dictionary protein classes
  • Assessment of the Genetic Effects of Polymorphisms in the . . .
    deposits 1 In the past, calcification of normal tissue has been recognized as a common component of atherosclerotic le-sions 2 Some evidence suggests that calcification of athero-sclerotic arteries is an organized, regulated process, rather than being a passive phenomenon of aging 3,4 Several factors related to bone and mineral formation have
  • Tissue expression of TNFRSF11A - The Human Protein Atlas
    Summary of TNFRSF11A (CD265, FEO, LOH18CR1, ODFR, PDB2, RANK, TRANCE-R) expression in human tissue Cytoplasmic and membranous expression in several tissues We use cookies to enhance the usability of our website If you continue, we'll assume that you are happy to receive all cookies IMMUNOHISTOCHEMISTRY DATA RELIABILITY; Data reliability
  • TNFRSF11A - Antibodies - The Human Protein Atlas
    TNFRSF11A (CD265, FEO, LOH18CR1, ODFR, PDB2, RANK, TRANCE-R) protein expression summary IHC N A WB PA N A ICC IHC N A WB N A PA IMMUNOCYTOCHEMISTRY i Validation i Supported i The subcellular location is supported by literature Immunofluorescent staining of human cell line A-431 shows localization to plasma membrane





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